Info cartes d'apprentissage

Ici, tu apprends tout sur cette carte d'apprentissage

P125moa



Cartes à choix multiple
Cartes normales
Total cartes
Catégorie
Langue
De
ADENOSINE (Adenocard) MOA
slows conduction AV node, interrupt re-entrant pathway neg. chronotrope acts onSA cells, 1st choice PSVT
TRANEXAMIC ACID (TXA, Cyklokapron) MOA
Reduces plasminogen activation, mitigating conversion to plasmin.
PROMETHAZINE (Phenergan) MOA
H receptor antagonist that blocks the actions of histamines by competitive antagonism at the H receptor. In addition to antihistamine effects, promethazine also possesses sedative, anti-motion, antiemetic, and considerable anticholinergic activity.
DILTIAZEM HCL (Cardizem) MOA
Block influx of calcium ions into cardiac muscle. Arterial and venous vasodilator. Reduces preload and afterload. Reduces myocardial oxygen demand.
METOPROLOL (Lopressor) MOA
Selective inhibitor of beta1-adrenergic receptors; completely blocks beta1 receptors, with little or no effect on beta 2 receptors at doses <100 mg;
ASPIRIN MOA
Prostaglandin inhibition Blocks Thromboxane A2 via COX 1. Vaso-Dil via the COX 2 pathway.
SUCCINYLCHOLINE (Anectine) MOA
Ultra short acting depolarizing skeletal relaxant that mimics acetylcholine as it binds with the cholinergic receptors on the motor end plate, producing a phase I block as manifested by fasciculations.
DIPHENHYDRAMINE (Benadryl) MOA
Blocks H receptors; decreases vasodilatation Reverses extrapyramidal reactions.
MANNITOL 20% MOA
Promotes the movement of fluid form the intracellular space to the extracellular space. Decreases cerebral edema and intracranial pressure. Promotes urinary excretion of toxins.
NITROPASTE MOA
Smooth muscle relaxant acting on vascular, bronchial, uterine and intestinal smooth muscle. Dilation of arterioles and veins in the periphery reduces preload and afterload, decreases the workload of the heart and, thereby, myocardial oxygen demand.
GLUCAGON beta blcker OD MOA
stabilize cardiac rhythm in beta-blocker overdose.
THIAMINE (Vitamin B1) MOA
Combines with ATP to form thiamine pyrophosphate coenzyme, a necessary component for carbohydrate metabolism. The brain is extremely sensitive to thiamine deficiency.
LORAZEPAM (Ativan) MOA
Anxiolytic, anticonvulsant, sedative; suppresses seizure produced by foci in cortex, thalamus and limbic areas.
HYDROCORTISONE/METHYLPREDNISOLONE (Solu-Cortef, Solu-Medrol) MOA
Suppresses acute and chronic inflammation Replaces absent glucocorticoids Immunosuppressive
NITROGLYCERIN MOA
Smooth muscle relaxant with dilation of arterioles and veins in the periphery, reduces preload and afterload, decreases the workload of the heart and, thereby, myocardial oxygen demand.
EPINEPHRINE allergic MOA
bronchial smooth muscle relaxation and dilation of skeletal vasculature Blocks histamine release.
IPRATROPIUM BROMIDE (Atrovent) MOA
Blocks acetylcholine at the parasympathetic sites in bronchial smooth muscle causing bronchodilation.
DOPAMINE (Inotropin) Andregic MOA
alpha 1 effects
PROCAINAMIDE (Pronestyl) MOA
Suppresses phase IV depolarization in normal ventricular muscle and Purkinje fibers, reducing automaticity of ectopic pacemakers; suppresses reentry dysrhythmias by slowing intraventricular conduction.
ATROPINE SULFATE MOA
inhibits action of acetylcholine at postganglionic parasympathetic neuroeffector sites.
ROCURONIUM (Zemuron) MOA
Rocuronium produces complete muscular relaxation by binding to the receptor for acetylcholine at the neuromuscular junction, without initiating depolarization of the muscle membrane. As the concentration of acetylcholine rises in the neuromuscular junction, Pancuronium is displaced and muscle tone is regained.
LIDOCAINE HCL (2%) MOA
Decreases automaticity by slowing the rate of Phase 4 depolarization.
HALOPERIDOL (Haldol) MOA
strong antidopaminergic and weak anticholinergic. Acts on the CNS to depress subcortical areas, midbrain, and ascending reticular activating system in the brain.
KETOROLAC (Toradol) MOA
anti-inflammatory acting non-narcotic analgesic activity by inhibiting prostaglandin synthesis.
PANCURONIUM (Pavulon) MOA
Pancuronium produces complete muscular relaxation by binding to the receptor for acetylcholine at the neuromuscular junction, without initiating depolarization of the muscle membrane. As the concentration of acetylcholine rises in the neuromuscular junction, Pancuronium is displaced and muscle tone is regained.
HEPARIN SODIUM MOA
IX, XI, XII, plasmin. Does not lyse existing clots.
TETRACAINE MOA
Blocks the initiation and conduction of nerve impulses
VERAPAMIL (Isoptin) MOA
Verapamil inhibits the movement of calcium ions across cell membranes. The slow calcium ion current blocked by verapamil is more important for the activity of the sinoatrial node and atrioventricular node than for many other tissues in the heart. Verapamil decreases atrial automaticity, reduces atrioventricular conduction velocity, and prolongs the atrioventricular nodal refractory period. In addition, verapamil depresses myocardial contractility, reduces vascular smooth muscle tone, and dilates coronary arteries and arterioles in normal and ischemic tissues.
RACEMIC EPINEPHRINE MOA
Stimulates alpha-1 receptors reducing subglottic edema in croup by mucosal vascular bed vasoconstriction. Stimulates beta-2 receptors in the smaller airways in lungs causing bronchodilation with relaxation of bronchial smooth muscles. Reduces airway resistance. Useful in treating laryngeal edema; inhibits histamine release.
MORPHINE SULFATE MOA
Alleviates pain through CNS actions Suppresses fear and anxiety centers in brain. Depresses brain stem respiratory centers. Decreases preload and afterload, decreasing myocardial oxygen demand.
AMIODARONE (Cordarone) MOA
Class III K channel blocker. Prolongation of Action Potential; non-competitive A and B sympathetic blocker; Ca,Na channel blocking effects.
SODIUM BICARBONATE 8.4% MOA
Reacts with hydrogen ions to form water and carbon dioxide
HYDROXOCOBALAMIN (Vitamin B 12) MOA
hydroxocobalamin binds cyanide ions to form cyanocobalamin
DIAZEPAM (Valium) seizure MOA
Potentates inhibitory neurotransmitters. Raises seizure threshold.Induces amnesia and sedation.
ALBUTEROL (Proventil, Ventolin) MOA
Selective b-2 agonist stimulates adrenergic receptors/smooth muscle relaxation, bronchdilator, peripheral vasodialtion.
OXYGEN MOA
Reverses hypoxemia.
FUROSEMIDE (Lasix) MOA
Inhibits electrolyte reabsorption promotes excretion of sodium, potassium, chloride
NOREPINEPHRINE (Levophed) MOA
Potent alpha-agonist resulting in intense peripheral vasoconstriction, positive chronotropic and increased inotropic effect with increased cardiac output. Alpha adrenergic activity resulting in peripheral vasoconstriction and beta-adrenergic activity leading to inotropic stimulation of the heart and coronary artery vasodilation.
AMYL NITRITE, SODIUM NITRITE, SODIUM THIOSULFATE (Cyanide Antidote Kit) MOA
produces thiocyanate, which is then excreted
ACETAMINOPHEN (Tylenol, Ofrimev) MOA
analgesics and antipyretics.
ACTIVATED CHARCOAL MOA
Adsorbs toxic GI
NALOXONE (Narcan) MOA
Competitive inhibition at narcotic receptor sites. Reverse respiratory depression secondary to depressant drugs. Completely inhibits the effect of morphine.
GLUCAGON hyperglcemia MOA
Increases blood glucose by stimulating glycolysis.
ONDANSETRON (Zofran) MOA
Selective 5-HT receptor antagonist, blocking serotonin, both peripherally on vagal nerve terminals and centrally in the CNS chemoreceptor trigger zone
DEXTROSE MOA
Rapidly increases serum glucose l
CALCIUM CHLORIDE / CALCIUM GLUCONATE MOA
inotrope May enhance vent automaticity. Helps stabilize the cell membrane
ETOMIDATE (Amidate) MOA
Short acting hypnotic of the reticular activating system
MEPERIDINE (Demerol) MOA
Synthetic opioid agonist that acts on opioid receptors to produce analgesia, euphoria, respiratory and physical depression; a schedule II drug with potential for physical dependency and abuse.
FENTANYL CITRATE (Sublimaze) MOA
narcotic analgesic
KETAMINE (Ketalar) RSI MOA
Blocks pain receptors and minimizes spinal cord activity, between the thalamus and the limbic system.
EPINEPHRINE cardiac MOA
+chrono, +inno,+dromo + automaticity.
KETAMINE (Ketalar) delerium MOA
Blocks pain receptors and minimizes spinal cord activity, between the thalamus and the limbic system.
DOPAMINE (Inotropin) dopaminergic MOA
dilate renal and splanchnic vasculature at low doses.
VASOPRESSIN (Pitressin) MOA
Vasopressin acts by direct stimulation of smooth muscle receptors. When given in extremely high doses, it acts as a noradrenergic peripheral vasoconstrictor.
DIAZEPAM (Valium)cardio MOA
Potentates inhibitory neurotransmitters. Raises seizure threshold.Induces amnesia and sedation.
HYDROMORPHONE (Dilaudid) MOA
synthetic analog morphine
FLUMAZENIL (Romazicon) MOA
Flumazenil antagonizes benzodiazepines in CNS. reverse sedation, impairment of recall, and psychomotor impairment produced by benzodiazepines. Will not reverse hypoventilation or antagonize CNS effects of ethanol, barbiturates, or opioids.
VECURONIUM (Norcuron) MOA
Vecuronium is an intermediate-acting, non-depolarizing, neuromuscular blocking agent. Non-depolarizing agents produce skeletal muscle paralysis by blockade at the myoneural junction. Unlike depolarizing agents, vecuronium has little agonist activity, with no depolarizing effect at the motor endplate.
GLUCOSE -ORAL MOA
fast glucose
PRALIDOXIME CHLORIDE (2-PAM, Protopam) MOA
Reactivation of cholinesterase to effectively act as an antidote to organophosphate pesticide poisoning. This action allows for destruction of accumulated acetylcholine at the neuromuscular junction.
DEXAMETHASONE (Decadron) MOA
surpress inflammation, immunosuppressive
DOPAMINE (Inotropin) Beta-andregenic MOA
+ inotropy. +chronotropy.
MIDAZOLAM (Versed) MOA
Increased production of GABA, an inhibitory neurotransmitter, causing procedural amnesia; anesthesia; skeletal muscle relaxation. Anxiolytic and sedative / hypnotic properties similar to other benzodiazepines.
KETAMINE (Ketalar) pain MOA
Blocks pain receptors and minimizes spinal cord activity, between the thalamus and the limbic system.
MAGNESIUM SULFATE MOA
Reduces striated muscle contractions and blocks peripheral neuromuscular transmission by reducing acetylcholinesterase release at the myoneural junction; manages seizures in toxemia of pregnancy; induces uterine relaxation; can cause bronchodilation after beta agonists and anticholinergics have been used.
© 2024 MemoCard